SCD vs semi-vegitarean mediterranean diet vs modulin IBD for Crohn's Disease

Abstract

The mediterranean diet and the Specific Carbohydrate Diet both have evidence of efficacy in ameliorating the symptoms of Crohn's Disease. However, there has not been a study of the efficacy of these diets given that the participants were not already being treated. This makes those studies practically meaningless, and instead of measuring the natural reaction of a subject to dietary change, they are dependent additionally on how diets may alter medication efficacy and how individuals react to medications. Th

Description

We expect from the association between NOD2 mutations and PAMP recognition that a significant depletion of intestinal gut bacteria will reduce inflammatory activity and consequent pathology in a Crohn’s Disease cohort. We alter the expected levels of signalling leading to inflammatory activity by altering the host diet, and by way of that, the host microbiome, to reduce the levels of muramyl dipeptide (the innate immunity target recognized by NOD2). We hypothesize we will see significant symptomatic improve

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SCD vs semi-vegitarean mediterranean diet vs modulin IBD for Crohn's Disease

Abstract

The mediterranean diet and the Specific Carbohydrate Diet both have evidence of efficacy in ameliorating the symptoms of Crohn's Disease. However, there has not been a study of the efficacy of these diets given that the participants were not already being treated. This makes those studies practically meaningless, and instead of measuring the natural reaction of a subject to dietary change, they are dependent additionally on how diets may alter medication efficacy and how individuals react to medications. Th

Description

We expect from the association between NOD2 mutations and PAMP recognition that a significant depletion of intestinal gut bacteria will reduce inflammatory activity and consequent pathology in a Crohn’s Disease cohort. We alter the expected levels of signalling leading to inflammatory activity by altering the host diet, and by way of that, the host microbiome, to reduce the levels of muramyl dipeptide (the innate immunity target recognized by NOD2). We hypothesize we will see significant symptomatic improve

View